Tuesday, February 8, 2011

Placenta Increta caused hemoperitoneum in the 26 weeks of pregnancy: a case report

Placenta Increta is a serious complication of pregnancy. We describe a case connected to uterine rupture with massive intra-abdominal bleeding.

A 34-year-old Caucasian Albanian women Gravida 2, paragraph 1, practiced the emergency room associated with our hospital for acute abdominal pain, a profound secondary anaemia. A placenta of Increta Destruens Anatomopathological diagnosis was made. A urgent hysterectomy after reanimation procedures due to the severe anaemia and the belly drama accompanying case applied performed. Saving intra-operatively, a uterus procedure was found to be impossible, and hysterectomy remained the only surgical option. The uterine structures were sent to the other microscopic evaluation. The histological investigation Trophoblastic was deep infiltration of the uterine wall observed the surgeon decision justify. Our patients received blood transfusions and antibiotics. Their threads are removed from the 8th postoperative day and was released the following day in a stable condition.

In this case, describe a patient with uterine rupture and massive bleeding, demonstrates a serious and potentially fatal complication of placenta praevia. In such cases, surgery is essential, and hysterectomy may be the only viable option.


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Saturday, February 5, 2011

Spinal arachnoid cysts break presentation with acute severe headaches secondary intracranial hypotension: a case report

Headache is a common presenting complaint and has a wide differential diagnosis. Clinicians must alert the clues that may suggest an underlying secondary etiology. We describe a new case of headache as secondary intracranial hypotension that arachnoid Cyst was precipitated by the fraction of a spine. Case a 51-year-old Indian female with sudden onset of severe headache suggestively presented a subarachnoid haemorrage. Inspections, including a computer tomography brain scans were normal cerebrospinal of fluid examination and a magnet resonance angiogram. Keep the headache and magnetic resonance imaging reveals bilateral thin subdural collections, a spinal subarachnoid cyst and a right-sided pleural effusion. This was in line with a diagnosis of headache as a secondary intracranial hypotension, as a result of spinal arachnoid cysts rupture.

A rare cause of spontaneous intracranial hypotension is spinal arachnoid cysts rupture. Spontaneous intracranial hypotension is still under diagnosed a heterogeneous condition. It should be daily appeared headache in the differential diagnosis of patients with new-onset function.


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Wednesday, February 2, 2011

Rapidly progressive polyneuropathy due to the dry beriberi in a man: a case report

We describe a case of rapidly progressive and seriously debilitating polyneuropathy in a patient with confirmed Hypovitaminosis B1, accordance with dry beriberi. The decisive factor was this is a treatable condition, although sometimes incomplete recovery, but it is probably under recognized yet increasingly faced with the increasing number of alcohol abuse in the Western world.

A 49-year-old Caucasian British man presented with progressive weakness of the lower extremities of about seven months duration. He known difficulty climbing stairs. He complained of also lethargy and the loss of muscle bulk, including his thighs. He had refused a history of type 2 approved but excessive alcohol consumption diabetes mellitus and previous alcohol abuse in the five years before the presentation. First clinical and Neurophysiological investigations were consistent with a mild peripheral neuropathy and probably proximal myopathy. However, in the subsequent four months he developed a marked quadriplegia with profound sensory disturbance of all players. Repeat neurophysiology revealed a widespread polyneuropathy with extensive acute and subacute denervation changes in all four limbs, and reduced or sensory nerves Aktionspotentiale absent. Malnutrition B1 has been confirmed (45nmol/L, reference area 66-200nmol/L). His rapid clinical deterioration was in accordance with dry beriberi. He was treated with thiamin. Subsequent follow-up but significant improvement revealed slowly, so by 15-16 months from the first onset of symptoms, and six months after the beginning of his marked quadriplegia, he was able, to stand independently and trust was gradually in walking up to a period of stationary Neurorehabilitation.

There is a potentially wide differential diagnosis for this type of presentation. Confirm Hypovitaminosis B1 by request of the assay before vitamin replacement guarantees accurate diagnosis and appropriate ongoing treatment. An increasingly high index is probably suspected in connection with the increasing number of alcohol abuse in the Western world and the increasing prevalence of dry beriberi possible needed.


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